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KMID : 0383819610080010085
Tuberculosis and Respiratory Diseases
1961 Volume.8 No. 1 p.85 ~ p.89
Adrenal Cortical Function in Pulmonary Tuberculosis
±è¼øÅÃ(ÑÑâ÷÷Ê)/Sun-Taek Kim
±è°æ½Ä(ÑÑÌ×ãÕ)/Kyung-sik Kim
Abstract
We measured total 17-Hydroxycorticosteroid excretion and its increasing rate after
ACTH administration by using Porter-Silber method and 17-ketosteroid excretion by
using Holtorff and Koch¢¥s method, and had measured reducing rate of circulating
eosinophile count after ACTH administration in order to investigate adrenal cortical
function of the pulmonary tuberculosis.
1) In Normal Control group.
17-Hydrokycorticosteroid excretion 4.91¡­7 93§·
average 5.46§·
Increasing ratio of 17-Hydroxycorticosteroid after
ACTH administration 30.89%
17-Ketoateroid excretion 13.66¡­23.91§·
average 17.71¢¥§·
Reducing ratio of eosinophile count
after AC7H administration 64%
In Minimal Tuterculosis group
17-Hydroxycorticosteroid excretion 1.93¡­4.30§·
average 3.19§·
17-Ketosteroid excretion 5.42¡­13.30§·
average 10.36§·
Reducing ratio of eosinophile count
after ACTH administration 44.83§·
In far advanced group
17-Hydrokycorticosteroid excretion 0.83¡­4.43§·
average 2.46§·
Increasing ratio of 17-Hydroxycorticosteroid
after ACTH administration 196.7%
17-Ketosteroid excretion 4.60¡­14.43§·
average 767§·
Reducing ratio of eosinophile count
after ACTH administration 52%
(8 hrs ¥°. ¥´. method)
2) The adrenal cortical function of the pulmonary tuberculosis depressed and its
degree was almost parallel with severity of the diseases.
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